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Recent News and Articles on the Keywords: executive + brain + 0.31  Related to the article below (Last Update: 8/5/2008)

Thallion Announces 2008 Second Quarter Results
Trading Markets (press release), CA - Jul 10, 2008
Net loss for the six-month period ended May 31, 2008 was $9917204 or 0.31 per share, compared to a net loss of $6577207 or $0.37 per share for the six-month ...TSE:TLN - OTC:CMTX

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Source: Google News

[PDF] Hypertension and the brain: vulnerability of the prefrontal regions and executive functions -
N Raz, KM Rodrigue, JD Acker - Behav Neurosci, 2003 - iog.wayne.edu
... and executive functions, which show significant age- related declines (Head & Raz,
2000; Salthouse, 1994). Fourth, only global indices of brain structural ...
-

Executive Functions Following Traumatic Brain Injury in Young Children: A Preliminary Analysis -
L Ewing-Cobbs, MR Prasad, SH Landry, L Kramer, R … - Developmental Neuropsychology, 2004 - Lawrence Earlbaum
... respectively, ? 2 (1, N = 83) = 0.31, p > .1 ... of general cognitive scores on the
executive function variables ... of variables are altered by brain injury, removing ...

… Children With Traumatic Brain Injuries: Assessment Using the Behavior Rating Inventory of Executive -
S Mangeot, K Armstrong, AN Colvin, KO Yeates, HG … - Child Neuropsychology, 2002 - informaworld.com
... Long-term deficits in executive functions following childhood traumatic brain injuries
(TBI) were examined using the Behavior Rating Inventory of Executive ...

… Skills, Emotional Status, and Executive Function in Psychosocial Adjustment Following Acquired Brain -
T Ownsworth, J Fleming - The Journal of Head Trauma Rehabilitation, 2005 - headtraumarehab.com
... model in the first step (R = 0.31, R 2 ... cognitive beliefs influence emotional adjustment
following brain injury.51 ... Metacognition and executive functioning TOP. ...

… general intellectual ability and performance on ecologically valid executive tests in a severe brain -
RL WOOD, C LIOSSI - Journal of the International Neuropsychological Society, 2006 - Cambridge Univ Press
... between performance on tests of executive function, controlling ... WAIS-III FSIQ, in
severely brain injured individuals ... Key Search 0.24** 0.47** 0.31** 0.26** 0.52 ...

Central executive system impairment in traumatic brain injury -
A Serino, E Ciaramelli, AD Santantonio, S Malag?, … - Brain Injury, 2006 - informaworld.com
... the relation between the severity of brain lesions and ... Executive functions 0.42 2.35
0.05 Working memory 0.69 3.4 ... ?1.82 0.08 Education 0.19 1.02 0.31 Time post ...

Executive function is associated with brain proton magnetic resonance spectroscopy in amnestic mild … -
HR Griffith - Journal of Clinical and Experimental Neuropsychology, 2006 - informaworld.com
... a lesser extent with the AVLT (range -0.31 to 0.46 ... Primarily, the relationship of
brain meta- bolic measures ... amnestic MCI to measures of executive function is ...

Recovery of executive skills following paediatric traumatic brain injury (TBI): A 2 year follow-up -
V Anderson, C Catroppa - Brain Injury, 2005 - informaworld.com
Page 1. ORIGINAL ARTICLE Recovery of executive skills following paediatric traumatic
brain injury (TBI): A 2 year follow-up V. ANDERSON & C. CATROPPA ...

Deep Brain Stimulation for Parkinson's Disease: Association between Stimulation Parameters and … -
P Francel, K Ryder, J Wetmore, A Stevens, K … - Stereotact Funct Neurosurg, 2004 - content.karger.com
... Bl GDS 0.31 0.23 0.55 0.21 ?0.13 0.42 ?0.33 ?0.02 ?0.14 ... P, Limousin-Dowsey P:
The impact of deep brain stimulation on executive function in ...

executive functioning: Improving planning skills in people with traumatic brain injury through the … -
J Hewitt, JJ Evans, B Dritschel - Neuropsychologia, 2006 - Elsevier
... impairment (but equally or more severe executive impairment ... steps in the plan (r =
0.28, p = 0.31). ... of a group of participants with traumatic brain injury, as ...

Source: Google Scholar

Older adults who develop deficits in executive function, which involves the brain's ability to problem-solve, make decisions and control negative thoughts, appears to lead to depression, researchers from Australia and the US report.

Lead author Dr. William von Hippel at the University of New South Wales in Sydney and his colleagues propose that deficients in executive function increase the risk of late-onset depression.

To test their theory, von Hippel's team recruited community-residing subjects between the ages of 66 and 92 years. The subjects were not demented and were not using psychoactive medications that could hinder cognitive function, according to the report, published in the journal of Cognitive Therapy and Research.

Twenty-four study participants reported depressive symptoms before the age of 60, and 20 patients had symptoms after the age of 60. They underwent a series of neuropsychological tests to measure ruminative thinking, depression, memory and cognitive function.

Analysis of the data showed that only the group with late-onset depressive symptoms had poor performance on tests of executive function. And only in the late-onset group was executive dysfunction positively correlated with rumination.

von Hippel and his colleagues suggest that age-related impairments in the brain's executive function prevent these individuals from regulating ruminative thoughts, which intensify their symptoms of depression.

Interventions to improve executive function or reduce ruminative thought patterns -- such as physical exercise, cognitive-behavioral or problem-solving therapies -- may prevent or improve symptoms of depression in later life, the researchers suggest.

However, they also note that larger, long-term studies will be required to firmly establish these relationships.

SOURCE: Cognitive Therapy and Research

Copyright © 2006 Reuters Limited. All rights reserved. Republication or redistribution of Reuters content, including by framing or similar means, is expressly prohibited without the prior written consent of Reuters. Reuters shall not be liable for any errors or delays in the content, or for any actions taken in reliance thereon. Reuters and the Reuters sphere logo are registered trademarks and trademarks of the Reuters group of companies around the world.

"It's not to say that people don't get over this, but...they're at greater lifelong risk, particularly if they develop dependence so rapidly that they have it this early in life," the study's lead author, Dr. Ralph W. Hingson of Boston University School of Public Health's Youth Alcohol Prevention Center, told Reuters Health.

There is mounting evidence that people who start drinking early are more likely to become alcohol dependent, Hingson and his team note in the Archives of Pediatrics and Adolescent Medicine. To investigate whether they may also become alcohol dependent at a younger age, the researchers analyzed the results of a 2001-2002 survey of 43,093 adults conducted by the National Institute on Alcohol Abuse and Alcoholism.

Twenty-seven percent of the men and women who started drinking before age 14 were alcohol dependent within 10 years, compared with 4 percent of those who began drinking at age 21 or older.

Using statistical techniques, the researchers factored in the influence of other factors that could be related to early drinking and the development of alcohol dependence, such as antisocial behavior during childhood, a family history of alcoholism, depression and education level.

Even after controlling for such factors, people who started drinking early were 2.6 times more likely to have episodes of alcohol dependence lasting longer than one year and nearly three times as likely to have six to seven symptoms of alcohol dependence versus three to five symptoms.

The findings underscore the dangers of early alcohol use, Hingson and his team note, and raise the possibility that efforts to help prevent drinking among teens, such as raising the drinking age to 21, could reduce the rates of alcohol dependence.

"We think it's very important that adolescents routinely be asked about their drinking practices by their health care providers," Hingson told Reuters Health. "There are interventions that we know can make a difference."

SOURCE: Archives of Pediatrics and Adolescent Medicine

Copyright © 2006 Reuters Limited. All rights reserved. Republication or redistribution of Reuters content, including by framing or similar means, is expressly prohibited without the prior written consent of Reuters. Reuters shall not be liable for any errors or delays in the content, or for any actions taken in reliance thereon. Reuters and the Reuters sphere logo are registered trademarks and trademarks of the Reuters group of companies around the world.

UV rays important to life, but also dangerous

Here comes the sun, Here comes the sun, And I say it's all right. ..."

Health advocates might tell the Beatles they needed to reword the pop tune that welcomes the summer.

Sun is all right, in moderation.

Sun makes ultraviolet rays, commonly called UV. And as much as we love our faces warmed by the sun, too much UV isn't all right - for our faces, eyes, arms, legs, hands, torsos, feet, even our toes.

"The No. 1 cause of premature aging and skin cancer is ultraviolet exposure," said Dr. J. Hunter Phillips III, a Biloxi, Miss., dermatologist. "That's the word I try to get out. Some listen; some don't. It's like smoking.

"I'd say that in the last decade, exposure to excessive UV has at least quadrupled due to more tanning beds. I also find the younger ones don't listen as much. They think they're invincible and there's a lot of peer pressure for a tan. The American Academy of Dermatology is trying to introduce legislation for tanning beds that would include informed consent and parental consent.

"People need to be aware that what they are doing now could be the cause later in life for skin problems and skin cancer."

Phillips and other dermatologists preach sunscreen, sunblock, protective clothing, hats, sunglasses and limited exposure.

"I'm a realist. You don't tell people not to go outside. I like being outside myself," Phillips said. "But everything in moderation. Go out early in the morning or late in the afternoon."

An Environmental Protection Agency Web page with a UV Index Forecast allows many Americans to see the ultraviolet potential for their communities. Recently, the Mississippi Coast was advised on the site developed by EPA and the National Weather Service (www.epa.gov/sunwise/uvindex.html) that:

"Protection against sun damage is needed. If you need to be outside during midday hours between 10 a.m. and 4 p.m., take steps to reduce sun exposure. A shirt, hat and sunscreen are a must, and be sure you seek shade. Beachgoers should know that white sand and other bright surfaces reflect UV and can double UV exposure."

If not computer savvy, you're still barraged with sun advice from advertisements and just about any medical professional.

Those with freckles, fair skin, or who are salon tanners or who are old-fashioned sun worshippers are duly warned. Beware of Mr. Sun and his ultraviolet rays.

We are a UV-aware society but whether we follow the advice is another story.

More than 10,000 Americans die each year from melanoma, and the cases get worse as the population's exposure to UV radiation appears to increase. The American Cancer Society expects 62,190 melanoma cases this year.

There are three types of UV: UVA (penetrates skin deeper and causes premature aging, cancer), UVB (causes sunburns and repeated exposure can cause cancer) and UVC (usually well filtered by ozone).

Layers of ozone shield the Earth from much of the sun's harmful UV radiation, but scientists say ozone depletion, along with seasonal and weather variations, cause different amounts of UV to reach the Earth at any given time.

And UV isn't all bad. Small amounts are essential for the production of vitamin D, although overexposure may result in acute and chronic, sometimes deadly health problems. As in so much we do for our health, it's a balancing act, one not always understood by the younger among us who put perceived attractiveness above sun warnings.

The World Health Organization jumped on the bandwagon this year when it advised no person younger than 18 should use a tanning bed. In reporting that exposure to UV, either naturally from the sun or from artificial sources such as sunlamps, is a known risk factor for skin cancer, WHO cited that one in three cancers diagnosed worldwide is a skin cancer.

---

3 TYPES OF UV SUN RAYS:

UVA

This one penetrates more deeply into the skin and is responsible for contributing to photodamage and wrinkling of the skin, premature aging and skin cancer. UVA is prevalent in tanning parlors where tanning devices can emit two to five times more UVA than natural sunlight.

UVB

These rays are shorter but stronger. UVB primarily affects the skin's outer layers and is thought to be the primary cause of sunburn, skin aging and skin cancer. UVB rays tend to be more intense during the summer, at higher altitudes, and at locales closer to the equator. Repeated exposure to UVB over the course of one's life can cause skin cancer and alter the immune system.

UVC

These are the strongest, most dangerous rays. However, little attention is given to UVC rays as they are normally filtered by the ozone layer and do not reach the Earth.

 
 
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